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Acta biochimica Polonica

doi: 10.1530/JOE-16-0598 [DOI] [PubMed] [Google Scholar] 37

Surprisingly, mice with specific genetic deletion of Gpx1 appear phenotypically normal and with normal life span (Ho et al., 1997), suggesting that there are alternative compensatory mechanisms for H 2 O 2 scavenging in Gpx1 deficiency

AIM2 suppresses ferroptosis by promoting proteasomal degradation of FOXO3a, a transcriptional activator of ACSL4 a key enzyme that esterifies PUFAs into peroxidation-prone phospholipids essential for ferroptosis execution (259)

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They concluded that the energy peripheral signals sensed to regulate fatty acid metabolism in the hypothalamus and consequently the feeding behavior, may not be a nutrient, but ghrelin through an action at the level of the VMH