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At physiological levels, ROS play an essential role in redox signaling by influencing the activity of transcription factors such as NF-B and Nrf2, and modulating pathways like MAPK and PI3K/Akt, which are vital for neuronal survival and adaptation (Finkel and Holbrook, 2000

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doi: 10.1038/ijo.2008.148 34 RegmiAAiharaEChristeMEVargaGBeyerTPRuanXet al

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Although tissue-specific differences in hepcidin sensitivity, along with variations in critical thresholds, time kinetics and the compensatory capability of cellular antioxidant defense systems, may modulate iron accumulation and ferroptotic outcomes, existing evidence supports a pro-ferroptotic role of hepcidin in Kupffer cells, neurons and hepatocytes (65-67)